INTRODUCTION:Fahr’s disease or Fahr’s syndrome is characterized by the calcification of basal ganglia and cerebral cortex, which is usually transmitted in an autosomal dominant manner. It is a rare neurological condition and most of the patients with the disease condition present with movement disorders, tremors, dyskinesia, and sometimes gait disturbances, cognitive impairment, speech disturbances, psychiatric changes such as mania, sensory changes or even pain [1-3]. Fahr’s disease should be differentiated from Fahr’s syndrome, which is usually secondary to underlying systemic pathology, this could be differentiated with blood tests for serum calcium, iPTH, along with other routine blood investigations. Moreover, basal ganglia calcification seen in CT scans could radiologically mimic the disease condition, thus clinical correlation with radiological investigations are the key in the diagnosis of Fahr’s disease [2, 4, 5]. Here, we present a case of a 65-year-old male, with Fahr’s syndrome secondary to hyperparathyroidism which initially mimicked as Parkinson’s disease and later improved after starting the patient on vitamin D and calcium carbonate therapy.
