Title:A 36-week preterm newborn with gastroschisis: A rare Case Report .Mouhammed Sleiay a,+, Bilal Sleiay a, Hasan Alsmoudi a, Mohammad ali abshia, Ahmad Buz a, Ahmed Merzaa, Hadi Alabdullah a, Mohammad Besher Fansa a, FERAS ALHARROUSH b.

Title: A 64 -year-old male with diffuse renal large B-cell non-Hodgkin lymphoma:A rare Case Report.Hasan Haydar a,+ ,Mouhammed Sleiaya, Hadi Alabdullaha , Rouba Al-alloush a, Nour Al_alloush a , Mohamad Yasin Lutfi b, Simon Youssefc, Suaad Hamshod.a Faculty of Medicine, Hama University, Hama, Syrian Arab Republic.b Hama National Hospital,Urology Department, Hama, Syrian Arab Republic.c Hama National Hospital,pathology Department, Hama, Syrian Arab Republic.d Rheumatology department Faculty of medicine, Damascus University, Damascus ,Syrian Arab Republic.*Corresponding author: Hasan Haydar, Faculty of Medicine, Hama University, Hama, Syrian Arab Republic. email: hasanhaydar458@gmail.com+ Hasan Haydar ,Mouhammed Sleiay, Rouba Al-alloush , Nour Al_alloush, Mohamad Yasin Lutfi , Simon Youssef , Suaad Hamsho are co-first authors. All contributed equally in this paper.Emails & ORCID:Hasan Haydar: hasanhaydar458@gmail.comMouhammed Sleiay:abdmouh1234mouhmouh@gmail.comORCID: 0009-0003-8887-0739Hadi Alabdullah :dr.alabdullah.md@gmail.comRouba Al-alloush : rouba2002alloush@gmail.comNour Al_alloush: nour2005alloush@gmail.comMohamad Yasin Lutfi: Doctor85yasen@gmail.comSimon Youssef:simon.youssef.1978@gmail.comSuaad Hamsho,soha.hamsho3@gmail.com

Abstract :Background: A noval radiologic sign in patients with renal failure and UE with metabolic acidosis has recently been identified as the lentiform fork sign. On magnetic resonance imaging (MRI), the ”lentiform fork sign” has been described as bilateral symmetrical hyperintensities in the basal ganglia encircled by a hyperintese rim delineating the lentiform nucleus. Changes in uremic solute retention, aberrant blood-brain barrier transport, disorderd vascular reactivity, altered electrolyte and acid-base balance, and altered hormone metabolism are the most likely causes of the condition.Case presentation : 56-year-old man with end-stage renal disease was brought to the emergency room for a progressive change in mental status and involuntary arm movements over the previous five days, which were also accompanied by mild dyspnea.A brain MRI was performed, and it revealed hyperintensity on T2/FLAIR in the white matter surrounding the basal ganglia. The haloperidol was stopped, and there more dialysis sessions were carried out.Conclusion : intensified hemodialysis and glycemic control are the cornerstones of treating DUS with likely reversible clinical symptoms and remission of imaging abnormalities.

Introduction:The term ”oxalosis” refers to the systemic buildup of calcium oxalate, the insoluble salt of oxalic acid, outside of the urinary system. Primary and secondary hyperoxalurias are the pathologic disease processes in charge of causing systemic oxalosis. The kidneys, bone, heart, blood arteries, and skin are major sites of oxalate accumulation that result in illness1. The primary hyperoxalurias are a class of uncommon autosomal recessive metabolic diseases characterised by abnormally excessive generation of serum oxalate and consequent tissue deposition. Most individuals first exhibit recurrent urolithiasis and renal failure when they are young. Primary hyperoxalurias are characterised by vascular deposition of skin manifestations that produce oxalate, such as livedo reticularis, acrocyanosis, peripheral gangrene, and ulcerations2. Skin symptoms, on the other hand, are uncommon in individuals who develop an oxalosis caused by renal insufficiency and, when they do, they are the consequence of extravascular deposition, resulting in calcified nodules and miliary papules3. Globally, it is estimated that 1 in 58,000 people suffer with primary hyperoxaluria. With Type 1 accounting for around 80% of cases, it is the most prevalent kind. Each of the two types accounts for around 10% of cases4. here we report a case of a 23 year-old female patient with a cutaneous oxalosis without livedo reticularis, acrocyanosis and ulceration, and it could be a very rare case in medical literature.