A document by Nabila Al-Tamimi. Click on the document to view its contents.

Vertebral Artery Occlusion with Ipsilateral Weakness due to Ischemic Stroke at the Medulla–Cervical Spinal Cord Junction: A Case ReportPeter Felton 1; Yugant Khand 2; Ram Saha 1Department of Neurology, Virginia Commonwealth University, Richmond, Virginia 23298-0599, USA.Nepalese Army Institute of Health Sciences - College of Medicine, Sanobharyang, Kathmandu, 44600 Nepal.AbstractPosterior circulation strokes can present with diverse neurological deficits, some of which challenge conventional localization principles. We report a case of a 74-year-old male with a history of prior right medullary infarct who presented with recurrent symptoms and was found to have a new medullary acute ischemic stroke in the setting of chronic right vertebral artery occlusion. Notably, the patient exhibited ipsilateral limb weakness, a finding attributable to involvement near the pyramidal decussation at the medulla–spinal cord junction. This case underscores the importance of detailed neuroanatomical understanding in vertebrobasilar ischemia and highlights the diagnostic complexity of brainstem strokes.

IntroductionThe raccoon (Procyon lotor (Linnaeus, 1758)) is native to North and Central America but has been introduced to numerous regions worldwide, where it is commonly maintained in zoological collections and increasingly as a companion animal. In Georgia, raccoons were introduced during the Soviet period and have since established local feral populations[1]. Highly adaptable, raccoons reproduce and disperse efficiently, particularly in riparian habitats such as those in the Lombardy region, where released individuals have become naturalised[2].Their introduction has had ecological, economic, and health implications. Native fauna sufer from predation, particularly nesting birds and amphibians, while agricultural and urban infrastructure are frequently damaged. Raccoons are also recognised reservoirs of zoonotic agents such as Baylisascaris procyonis , rabies virus, canine distemper virus, Trypanosoma cruzi , and agents of mange and histoplasmosis. Despite these risks, no structured management strategy for feral raccoon populations exists in Georgia[2].Ovariectomy (OVE) is a routine sterilisation procedure in domestic dogs and cats, particularly in Europe[3]. Laparoscopic OVE (lapOVE) has been well documented in canines[4, 5] and offers distinct advantages over open surgery, including reduced tissue trauma, postoperative pain, and recovery time, along with enhanced visualisation[6].However, laparoscopic sterilisation techniques have not been described in raccoons. The present report details the first successful two-port laparoscopic ovariectomy in a raccoon, highlighting the anaesthetic protocol, surgical approach, and postoperative outcome.

Congenital Lobar Emphysema in an Infant Presenting with Persistent Cough and Respiratory Distress: A Case Report

1. INTRODUCTIONRetinal dysplasia (RD) is defined as the abnormal differentiation of the retina.1-5 True RD may result from failure of the inner and outer layers of the optic cup to be in contact during embryogenesis, resulting in retinal detachment.6-8 In dogs, the most common RD form is characterized by multifocal folds and rosettes in retinas that are otherwise mostly attached to the underlying retinal pigment epithelium.9,10 Larger RD lesions are defined as geographic and may develop in regions of partial retinal detachment.4,11,12 Dysplastic changes typically develop in the canine retina at 45-50 days of gestation and generally can be diagnosed before 10 weeks of age, when the retinal layers are fully developed.1-11Retinal dysplasia can be inherited or acquired. For example, Labrador Retrievers and Samoyeds may be affected by oculoskeletal dysplasia (OSD) or dwarfism associated with RD (drd).13,14 This condition is caused by mutations in either COL9A3 (drd1 in the Labrador Retriever) or COL9A2 (drd2 in the Samoyed).13,14Geographic RD-like lesions that develop later than 10 weeks of age were described in some canine breeds, such as Golden Retrievers, German Shepherds, and Labrador Retrievers.11,13 By routine ophthalmoscopy, these cannot be differentiated from true geographic RD, and they are being diagnosed as such.15,16 However, high-resolution imaging in affected Labrador Retrievers, Golden Retrievers, and German Shepherds revealed that at least some of these geographic RD-like lesions consist of inner retinal plaques with tubular rosettes and vesicle-like structures.11,13,17,18 The pathogenesis of these retinal plaques has not been defined; they may be acquired, but a genetic predisposition has not been ruled out.Geographic RD is recognized as an inherited condition in the Cavalier King Charles Spaniel (CKCS).15,16 The breeding recommendations by the Genetics Committee of the American College of Veterinary Ophthalmologists (ACVO) for RD-affected CKCS are similar to most other canine breeds: While dogs with geographic RD should not be bred, dogs with focal/multifocal RD or retinal folds can still be bred (‘breeder option’).15 The Hereditary Eye Diseases (HED) Committee of the European College of Veterinary Ophthalmologists (ECVO) states that breeding is optional for dogs affected by focal/multifocal or geographic RD unless the breed club issues different advice.16 According to the Blue Book on Ocular Disorders Presumed to Be Inherited in Purebred Dogs, published by the ACVO Genetics Committee, of the 17,092 CKCS examined between 2019 and 2023, 1.6% had geographic RD, while 3.2% had retinal folds.15 Even though these prevalences are higher than in the Labrador Retriever with 0.4% geographic RD and 0.9% retinal folds,15 no detailed morphological and clinical reports on geographic RD in the CKCS have been published to the best of our knowledge. It is unknown if these lesions represent true RD or are similar to the inner retinal plaques that develop later than 10 weeks of age in Labrador Retrievers, Golden Retrievers, and German Shepherds.11,13,17,18 To fill this knowledge gap, our retrospective study aims to describe high-resolution imaging of geographic RD in a CKCS using confocal scanning laser ophthalmoscopy (cSLO), optical coherence tomography (OCT), and OCT angiography (OCTA). We compared our findings with the images of a Labrador Retriever with unilateral inner retinal plaques, since this condition has been previously characterized.11,13,17,18

Escherichia coli–Associated Infectious Aortitis Complicated by Abdominal Aortic Aneurysm and Diverticulitis Treated with Covered Endovascular Reconstruction of the Aortic BifurcationChing-Chou Pai 1, Ya-Ting Chuang2, Hua-Yu Lin2, Tao-An Chen3,4,*Department of Cardiovascular Surgery, Show Chwan Memorial Hospital, Changhua 500, Taiwan.Surgical Intensive Care Unit, Department of Nursing, Show Chwan Memorial Hospital, Changhua 500, Taiwan.Division of Respiratory Therapy, Department of Chest Medicine, Show Chwan Memorial Hospital, Changhua 500, Taiwan.Institute of Emergency and Critical Care Medicine, National Yang Ming Chiao Tung University, Taipei City 112304, Taiwan* Corresponding authorTao-An ChenDivision of Respiratory Therapy, Department of Chest Medicine, Show Chwan Memorial Hospital,No. 542, Section 1, Zhongshan Road, Changhua City, Changhua County 50009, TaiwanPhone: +886-4-725-6166 ext. 81350Email: B117100045@tmu.edu.twORCID: https://orcid.org/0000-0002-0740-0619Key word : Aortitis, Aortic Aneurysm, Abdominal, Mycotic Aneurysm, Escherichia coli , Bacteremia, Endovascular Procedures

Severe pharyngo-laryngeal mucositis during radiotherapy in a patient receiving colchicine: a case report

Correction to ‘Treatment of Recurrent IgA Nephropathy After Kidney Transplantation: Case Report and Comprehensive Literature Review’A. Matarneh, O. Salameh, M. Koirala, et al., “ Treatment of Recurrent IgA Nephropathy After Kidney Transplantation: Case Report and Comprehensive Literature Review,” Clinical Case Reports 13, no. 11 (2025): e71474, https://doi.org/10.1002/ccr3.71474.In the published version of this article, errors were identified in the reference list limited to bibliographic citation details, including incorrect or incomplete author names, journal titles, publication years, volume or issue numbers, and page ranges. During the correction process, the reference list was reviewed against the original source publications and corrected where necessary to ensure accuracy and consistency with the cited literature. No references were added or removed, and no changes were made to the in-text citations, scientific content, data interpretation, or conclusions of the article. The authors apologize for these errors.Below are references that needs to be corrected, mistake that have been noted are pertaining to DOI’s linking and names.References #6In Published versionC. Jäger, C. S. Wang, T. Keller, et al., “Recurrence of IgA Nephropathy After Kidney Transplantation,” BMC Nephrology  23  (2022): 164, https://doi.org/10.1186/s12882-022-02802-x.PubMed link incorrect, other links are correct.Correct PubMed:https://pubmed.ncbi.nlm.nih.gov/35538438/Jäger C, Stampf S, Molyneux K, Barratt J, Golshayan D, Hadaya K, Huynh-Do U, Binet FI, Mueller TF, Koller M, Kim MJ. Recurrence of IgA nephropathy after kidney transplantation: experience from the Swiss transplant cohort study. BMC nephrology. 2022 May 10;23(1):178.Reference #7In Published versionN. Leeaphorn, N. Garg, E. V. Khankin, and F. Cardarelli, “Recurrence of IgA Nephropathy After Kidney Transplantation Among the US Population,” Transplantation Direct  3 , no. 4 (2017): e144, https://doi.org/10.1097/TXD.0000000000000642.In new versionLeeaphorn N, Garg N, Khankin EV, Cardarelli F, Pavlakis M. Recurrence of IgA nephropathy after kidney transplantation in steroid continuation versus early steroid‐withdrawal regimens: a retrospective analysis of the UNOS/OPTN database. Transplant International. 2018 Feb;31(2):175-86.Reference #9In Published versionT. Moriyama, K. Nitta, K. Suzuki, et al., “Latent IgA Deposition From Donor Kidney Is a Major Risk Factor for Recurrent IgA Nephropathy,” Clinical Transplantation  19 , no. Suppl 14 (2005): 41–47, https://doi.org/10.1111/j.1399-0012.2005.00446.x.DOI is incorrect.In new versionThe correct DOIDOI: 10.1111/j.1399-0012.2005.00403.xMoriyama T, Nitta K, Suzuki K, Honda K, Horita S, Uchida K, Yumura W, Tanabe K, Toma H, Nihei H, Yamaguchi Y. Latent IgA deposition from donor kidney is the major risk factor for recurrent IgA nephropathy in renal transplantation. Clinical transplantation. 2005 Aug;19:41-8.Reference #10In Published versionD. Maixnerová, A. Krajčovičová, J. Skibová, et al., “Outcome of 313 Czech Patients With IgA Nephropathy After Kidney Transplantation,” Kidney International Reports  6 , no. 9 (2021): 2315–2327, https://doi.org/10.1016/j.ekir.2021.06.024.In new versionMaixnerova D, Hruba P, Neprasova M, Bednarova K, Slatinska J, Suchanek M, Kollar M, Novak J, Tesar V, Viklicky O. Outcome of 313 Czech patients with IgA nephropathy after renal transplantation. Frontiers in Immunology. 2021 Sep 30;12:726215.Reference #12In Published versionJ. Floege, “Recurrent IgA Nephropathy After Renal Transplantation,” Seminars in Nephrology  24 , no. 3 (2004): 287–291, https://doi.org/10.1016/j.semnephrol.2004.03.008.In new versionThe correct DOIDOI: 10.1016/j.semnephrol.2004.01.008Floege J. Recurrent IgA nephropathy after renal transplantation. InSeminars in nephrology 2004 May 1 (Vol. 24, No. 3, pp. 287-291). WB Saunders.Reference #18In Published versionB. H. Rovin, H. J. L. Heerspink, J. Barratt, et al., “Efficacy and Safety of Sparsentan Versus Irbesartan in Patients With IgA Nephropathy (PROTECT): 2-Year Results From a Randomised, Active-Controlled, Phase 3 Trial,” Lancet  402 , no. 10416 (2023): 2034–2048, https://doi.org/10.1016/S0140-6736(23)02088-7.In new versiondoi: 10.1016/S0140-6736(23)02302-4. Epub 2023 Nov 3Rovin BH, Barratt J, Heerspink HJ, Alpers CE, Bieler S, Chae DW, Diva UA, Floege J, Gesualdo L, Inrig JK, Kohan DE. Efficacy and safety of sparsentan versus irbesartan in patients with IgA nephropathy (PROTECT): 2-year results from a randomised, active-controlled, phase 3 trial. The Lancet. 2023 Dec 2;402(10417):2077-90.Reference #19In Published versionH. J. L. Heerspink, V. Perkovic, R. M. Perkins, et al., “Early Proteinuria Reduction by Sparsentan Predicts Long-Term Kidney Outcomes in IgA Nephropathy: Interim Results From PROTECT,” Lancet  401 , no. 10386 (2023): 95–106, https://doi.org/10.1016/S0140-6736(22)02203-3.In new versionHeerspink HJ, Tesar V, Komers R, Hendry BM, Preciado P, Murphy E, Rovin BH. Implications of Proteinuria Remission on Estimated Glomerular Filtration Rate (eGFR) Trajectory in Patients with IgA Nephropathy in the PROTECT Trial: FR-PO872. Journal of the American Society of Nephrology. 2024 Oct 1;35(10S):10-681.

A document by ahmad matarneh. Click on the document to view its contents.

Correction to ‘Spurious Hyperphosphatemia From Alteplase-Treated Catheter: A Case Report and Literature Review’A. Matarneh, S. Sardar, O. Salameh, et al., “ Spurious Hyperphosphatemia From Alteplase-Treated Catheter: A Case Report and Literature Review,” Clinical Case Reports  13, no. 8 (2025): e70665, https://doi.org/10.1002/ccr3.70665.In the published version of this article, errors were identified in the reference list limited to bibliographic citation details, including incorrect or incomplete author names, journal titles, publication years, volume or issue numbers, and page ranges. During the correction process, the entire reference list was reviewed against the original source publications and corrected where necessary to ensure accuracy and consistency with the cited literature. No references were added or removed, and no changes were made to the in-text citations, scientific content, data interpretation, or conclusions of the article. The authors apologize for these errorsReference #9In Published paperA. M. Ball, W. D. Tobler, L. D. Ferrell, and L. Chan, “Spurious Hyperphosphatemia due to Sample Contamination With Heparin From Arterial Catheters,” American Journal of Kidney Diseases  44 , no. 2 (2004): 367–371, https://doi.org/10.1053/j.ajkd.2004.04.033.In new versionSchiller B, Virk B, Blair M, Wong A, Moran J. Spurious hyperphosphatemia in patients on hemodialysis with catheters. American journal of kidney diseases. 2008 Sep 1;52(3):617-20.Reference #14In Published paperT. Sav, H. Yilmaz, A. V. Kara, E. Memisoglu, and O. Cakmak Oksuz, “Spurious Hyperphosphatemia in Hemodialysis Patients: Effect of Catheter-Locking Solutions,” Hemodialysis International  19 , no. 2 (2015): 317–320, https://doi.org/10.1111/hdi.12250.In new versionSombolos K, Bamichas G, Hatsiou V, Fragidis S, Rizos A, Natse T, Fytili C. Alteplase as hemodialysis catheter locking solution and spurious hyperphosphatemia. The Journal of Vascular Access. 2011 Jul;12(3):269-.

Hypokalemic Periodic Paralysis in a Young Woman with Mast Cell Activation Syndrome: Atypical Presentation Associated with an Ultra-Rare CACNA1S VariantAli Moradi, Yasmin Aboutaleb, Saba Noreen, Abdullah Sahyouni, Hasin Sharma, Olugbenga Oyesanmi, Ian M. Kahane, Yizhi Lin, Hakan R. Toka1Ali Moradi M.D. Internal Medicine Resident, HCA Florida Blake Hospital, University of South Florida (USF), Morsani College of Medicine, Bradenton, USA. Email: Ali.moradi@hcahealthcare.com2Yasmin Aboutaleb M.D. Internal Medicine Resident, HCA Florida Blake Hospital, University of South Florida (USF), Morsani College of Medicine, Bradenton, USA. Email: Yasmin.aboutaleb@hcahealthcare.com3Saba Noreen, M.D. Internal Medicine Resident, HCA Florida Blake Hospital, University of South Florida (USF), Morsani College of Medicine, Bradenton, USA. Email: Saba.Noreen@hcahealthcare.com4Abdullah Sahyouni, M.D. Internal Medicine Resident, HCA Florida Blake Hospital, University of South Florida (USF), Morsani College of Medicine, Bradenton, USA. Email: Abdullah.Sahyouni@hcahealthcare.com5Hasin Sharma, Medical Student, Nova Southeastern University Kiran C. Patel College of Osteopathic Medicine, Florida, USA. Email: Hs987@mynsunova.edu6Olugbenga Oyesanmi, M.D. MPH, Division Research Director, HCA Florida Blake Hospital, University of South Florida (USF), Morsani College of Medicine, Bradenton, USA Email: Olugbenga.Oyesanmi@hcahealthcare.com7Ian M. Kahane M.D., Attending Physician, Internal Medicine, HCA Florida Blake Hospital, University of South Florida (USF), Morsani College of Medicine, Bradenton, USA. Email: Ian.Kahane@gmail.com8Yizhi Lin DO, Program Director, Internal Medicine, HCA Florida Blake Hospital, University of South Florida (USF), Morsani College of Medicine, Bradenton, USA. Email: Yizhi.Lin@hcahealthcare.com9Hakan R. Toka, M.D, PhD, Attending Physician, Nephrologist, Manatee Kidney Diseases Consultants, Bradenton, USA. Email: Hrtoka@gmail.com

Article title: Intraluminal hair in perforated appendicitis

Title : Severe Bilateral Sudden Sensorineural Hearing Loss Following Mild COVID-19 Infection Requiring Cochlear Implantation: A Case ReportTena Šimunjak, MD1,, Luka Županović, MD1, Filip Bacan, MD2, Andro Košec, MD, PhD2,3, Jakov Ajduk, MD, PhD2,31Department of Otorhinolaryngology, Head and Neck Surgery, University Hospital Sveti Duh,Zagreb, Croatia.2Department of Otorhinolaryngology & Head and Neck Surgery, University Hospital Center Sestre Milosrdnice, Zagreb, Croatia.3School of Medicine, University of Zagreb, Zagreb, Croatia.Corresponding Author:Tena Šimunjak,MDDepartment of Otorhinolaryngology, Head and Neck Surgery, University Hospital Sveti Duh,Zagreb, CroatiaUlica Sveti Duh 64, 10 000 Zagreb, CroatiaTel:+385989584948E-mail:tsimunjak@kbsd.hrWritten informed consent was obtained from the patient and the study was prepared according to the CARE case report guidelines.

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Key Clinical Message:Tumor-induced osteomalacia should be considered in patients with unexplained bone pain and fractures. Advanced imaging, such as Ga68-DOTATATE PET/CT, enables precise tumor localization, and surgical excision can rapidly normalize phosphate metabolism and resolve symptoms, underscoring the importance of early diagnosis and intervention.Keywords: Oncogenic osteomalacia, Tumor-induced osteomalacia, Fibroblast growth factor 23, Paraneoplastic Syndromes

A document by Shifeng Lin. Click on the document to view its contents.

Correction to “Thymic Neuroendocrine Tumor Presenting with Cervical Lymphadenopathy: A Case Report and Diagnostic Challenge ”X. Xie, J. Tang, W. Wang, D. Yang, and K. Qin, “ Thymic Neuroendocrine Tumor Presenting With Cervical Lymphadenopathy: A Case Report and Diagnostic Challenge,” Clinical Case Reports  13, no. 12 (2025): e71580, https://doi.org/10.1002/ccr3.71580.The order of the affiliations to article being corrected.The order of the institutions in the ”Author Affiliations” section of this article is incorrect. The correct order of the institutions should be as follows:Second Department of Thoracic Surgery, Hunan Cancer Hospital/The Affiliated Cancer Hospital of Xiangya School of Medicine, Central South University Changsha China.Department of Cardiovascular Surgery, The Second Xiangya Hospital, Central South University, 139 Middle Renmin Road, Changsha 410011, ChinaWe apologize for this error.

Precision under Pressure: Scalp Block and Dexmedetomidine for Brain Abscess Drainage in a Patient with Complex Cyanotic Heart Disease.Sandeep Khatri 1 | Ashish Raj Tiwari1 | Barsha Karki1| Manisha Poudel1 | Anish Sah1