not-yet-known not-yet-known not-yet-known unknown INTRODUCTION Wallenberg syndrome, initially documented in 1808 by Gaspard Vieusseux, received a more comprehensive delineation in 1895 from Adolf Wallenberg. He characterized it as an infarction that occurs in the lateral medulla oblongata due to occlusion of either the vertebral artery (VA) or the posterior inferior cerebellar artery (PICA). This syndrome is also known as lateral medullary syndrome (LMS) or posterior inferior cerebellar artery syndrome(1). looms as the leading brain stem ischemic stroke syndrome, although less frequent in occurrence compared to other stroke types. Among the cases of LMS, 75% are due to atherothrombotic events in large arteries, 17% originate from cardioembolism, and the remaining 8% arise due to vertebral artery dissection(2). The hallmark symptoms of LMS comprise pain and loss of temperature on the ipsilateral face and contralateral body, ipsilateral ataxia, vertigo, nystagmus, dysphagia, hoarseness, hiccups, and Horner’s syndrome(3). The clinical recognition of patients with LMS is of particular importance due to its association with vertebral artery dissection in 15 to 26% of cases and the favorable prognosis associated with optimal management(4). Brain magnetic resonance imaging serves as the cornerstone for diagnosis. Early recognition and the initiation of treatment lead to a favorable prognosis for this syndrome(5). We describe the case of a 42-year-old woman with LMS. Despite not receiving thrombolytic treatment, she experienced gradual recovery and achieved functional independence.