IntroductionThe spinal cord is supplied by three longitudinal arteries: One anterior spinal artery and two posterior spinal arteries with an extensive collateral network making the spinal cord very resistant to ischemia. Hence spinal cord infarction is a rare occurrence, accounting for only approximately 1 % of alle strokes [1], and usually involves the anterior spinal artery [2]. Because of the paired blood supply from the posterior spinal arteries, infarction here is uncommon and estimated to be 11 % of spontaneous spinal cord infarctions [3]. The leading cause of spinal cord infarction is aortic surgery followed by aortic dissection and aneurism, but in 21 % no established etiology is found [4]. The clinical neurological deficits of the spinal cord is linked to the affected anatomical territory: For instance, the anterior spinal cord artery supplies the anterior two-thirds of the spinal cord, and anterior spinal cord infarction therefore results in palsy, deficit of thermal sensitivity with relative retained sense of position and vibration (due to sparing of the dorsal columns) [5].Brown-Sequard syndrome is a hemicord syndrome resulting in disruption of the:Corticospinal tract resulting in ipsilateral weaknessDorsal column resulting in ipsilateral loss of proprioceptionSpinothalamic tract resulting in contralateral loss of pain and temperature sensationThe leading cause of Brown-Sequard syndrome is traumatic, but other causes include vertebral disc herniation, tumors, radiation, hemorrhage, and cervical spondylosis [6].We report a case of spontaneous posterior cord infarction resulting in a Brown-Sequard syndrome.