Through observation and experimentation of some individuals who suffering from

genetic tooth decay, it was found that decay is more active when eating some foods

containing triglycerides. Caries is transmitted between the teeth of the mills during

energy emission due to initial conditions such as genetic factors, and therefore caries

passes as an infection. Necrosis is increased after the use tooth-sticks, especially when

entering the air, which increases the reaction of combustion of the soft layer. The

prevalence of caries depends on the absorption of the entropy according to resonance

frequency. Moreover, dental necrosis is a dynamic system.

Introduction

This brief article discusses the mechanism of tooth decay that occurs in some people who

have a genetic factor, according to experience and observation. "To date, mutations in

the gene encoding amelogenin have been shown to underlie a subset of the X-linked

recessive forms of AI" [1]. Since decay affects the individual and settles in trouble, it

stimulates us to search for causes, to know the actual causes of tooth decay or factors

that help to stimulate tooth decay and the appearance of necrosis.

The aim is to produce an effective treatment for the elimination of the disease. Thus,

there are many questions about dental caries and feeling pain when eating a particular

food, and there is a relationship between food and caries activity, as eating sugar did not

affect, so the saying is known: "to activate the bacteria decay when there is energy, such

as sugar" has become unconvincing.

Why is dental pulque formed immediately after use of cleaning tooth-stick?

Why do we feel toothache especially those exposed to decay, after eating specific foods?

Why do necrosis occur horizontally between the teeth of the mills, where the infection of

necrosis is passed to the corresponding tooth on the other side of the mouth?

How to stimulate secretion cells that secrete mineralization proteins to induce analogue

synthesis to restore tooth necrosis structure?

Hypothesis

1. The mills teeth are covered by periosteum-like membrane that plays a catalytic role in

the growth of damaged cells, and therefore, dental enamel cells grow slowly, which

starting with a soft fibrous layer, and ending with mineralization.

2. Tooth decay occurs through the combustion reaction that occurs between stearin and

oxygen in soft layer, when there are cracks between the gums and teeth, with the

auxiliary factor of both genetics and oral microbes.

3. The tooth necrosis infection is transmitted through the emission of resonance

frequency, according to the corresponding function, and therefore the necrosis occurs in

the tooth corresponding to the emitter tooth if the auxiliary factors are available.

Methods and Materials

Through the experience in giving some people, specifically those who have a hereditary factor in tooth decay. Where they gave pure wheat food cooked with palm oil as well as

eating dates, sugar and chocolate containing bovine fat (triglycerides), as well as those

who use the tooth-stick of some plants to clean their teeth, after chewing the khat leaves.

Results and Conclusions

When they eat food cooked with palm oil, found that they suffer from pain in the areas of

necrosis (tooth decay), also when eating some dates, they feel severe pain in the teeth,

especially in areas where tooth decay has spread, as well as in some areas of the teeth, in

which no necrosis has yet appeared, and after a while, the necrosis begins to appear

slowly until it erodes the enamel area of the teeth, and therefore it is possible to identify

areas that are threatened with tooth decay (e.g., when feeling pain before black spots

appear on the teeth) and thus avoid this. In anther words, when the temperature of that

part of the mouth rises (you may feel this when you put the head of the tongue where the

pain is), then the stearin combustion reaction (thermal transformation as a physical

change) occurs, which leading to tooth necrosis, which results in dental necrosis

(chemical change). When eating vegetable sugar, (although there is decay), it does not

affect as strongly as the effect of eating a piece of chocolate containing bovine fat

(triglycerides) or eating dates. There is a mechanism to break down the molecular bonds

that make up the teeth, and thus return them into necrosis (the transformation of raw

materials into another as a result of thermal combustion). Therefore, the closest

meaning is that these chemical and physical processes occur in the frost of the fire phase,

as well as saliva at the front of the tongue plays an active role for tooth decay and

specifically molars.

Therefore, tooth decay occurs as a result of the combustion interaction between stearin

(as triglycerides), and oxygen, with the help of both genetics and oral microbes. “POs and

PKOo are both products of the palm oil industry that presently have limited use” [2]. As

found in the experiment, when eating only bread from brown wheat, and Palm oil, there

is pain occurs in the teeth resulting from the activity of caries.

In addition, the presence of cracks between the tooth and gums as a result of the

introduction of tooth-stick to remove the remnants of food is the starting point for the

onset and appearance of caries (tooth decay), and when eating dates palm, pain occurs in

the teeth because of the presence of palm stearin. As shown by some during the activity

of caries accompanied with pain, the air intake of the mouth also increases the pain,

which means, stimulate the combustion process through the availability of oxygen. Thus,

oxygen entering the mouth, and because of the presence of cracks between the teeth and

stearin, it leads to an interaction that turns the stearin into anther phase and then

combustion the surface of the teeth slowly, leaving the black necrosis. Although the

genetic factor plays a role in creating an environment for decay. And the cracks between

the teeth play an auxiliary role in tooth decay, leaving the food residues that are difficult

to remove, especially the remnants of khat (which chewed here in Yemen), inhibits the

combustion reaction to tooth decay by preventing the entry of oxygen, thus preventing

the combustion reaction, while the use of tooth-stick to extract food residue from the

teeth increases tooth decay, this does not mean neglecting the cleanliness of the teeth,

but rather the correct use of fine threads to extract food from the teeth. Therefore, the

best way to clean the teeth without exposing the surfaces of teeth scratches is using a

piece of cotton or finger.

Since exposure to smells that carry particles produced by food cooked with palm oil

(which contains stearin) leads to burning the mouth of the stomach and discouraging

appetite as well. Thus, the reaction takes its energy from the tooth enamel and therefore

necrosis continues until it includes most of the tooth, which is just carbon due to the

combustion reaction of the dental components. dental components.

This causes burning of the outer surface of the teeth (enamel teeth) and turning it into

necrosis (carbon). “Transmission electron micrographs of individual mineralized

collagen fibrils show that hydroxyapatite crystals are located mainly within the fibrils at

the level of the gap regions” [3]. Moreover, that after reaching the denture, the

combustion reaction occurs spontaneously when oxygen enters, which interacts with a

certain stearin of the collagen. Therefore, palmitic acid (in palm oil) arises and reacts

with oxygen and oral microbes and turns into heat combustion which causes necrosis, as

well as damage bones, heartburn, and necrosis of the brain. Acidic phospholipids and

specific proteolipids present in cell membranes play a key role in microbial

mineralization [4]. Moreover, the presence of proteins in saliva, denture or dental

plaque, all of which contain triglycerides, plays a role in tooth decay and necrosis, as well

as in the deposition of lime on the teeth (e.g., the presence of yellow pigment on the teeth

when the stearin is exposed to oxygen). In addition, there is a gene consisting of tooth

decay coding. Therefore, deposition of minerals by mouth microbes is also exposed to

combustion reaction due to the reaction of the stearin with oxygen and thus the

formation plaque.

“Enamelins are a major group of 28-70-kDa acidic proteins rich in aspartic acid,

glutamic acid, serine, and glycine found in developing and mature extracellular enamel;

a unique and highly mineralized ectodermal tissue covering vertebrate teeth” [5]. “The

nail is a specialized keratinous skin appendage that grows approximately 2 to 3 mm per

month, with complete replacement achieved in 6 to 9 months” [6]. “Acidic phospholipids

and specific proteolipids present in cell membranes play a key role in microbial

mineralization” [7].

Since the teeth contain a quantity of keratin, and therefore the teeth of the mills grow

very slowly without feeling them if and only if one of them is exposed to corrosion. This

is evident in the person who eats khat, when chewing khat for 8 hours a day, his teeth are

significantly eroded, and when the person changes the place of chewing (from the right

side to the left), the teeth that chewed khat have grown again. Therefore, the presence of

an membrane-like periosteum between the teeth (activated by electrochemical signals)

plays a catalytic role in re-growth of enamel cells. As a result, it is necrosis because cell

division makes the structure soft, and when stearin interacts with oxygen, decay and

necrosis occur. Therefore, when the membrane layer is scratched, it stimulates cell

growth to repair the corroded layer. While it starts with a fibrous layer before it becomes

mineralized, it is susceptible to plaque and to a combustion reaction if oxygen enters and

interacts with stearin during fat-containing meals Triple.

In addition, the acquisition of vibrational energy, which leads to the accumulation of

entropy of the teeth according to the effect of electromagnetism by the force applied

(stearin) on the molecules and enough to emit the decay particles (energy of necrosis'

entropy) in a straight line. Therefore, the enthalpy of the reaction, which are absorbed by

the material, are the stearin which reacts within the molecules. If a certain frequency

corresponds to the resonance frequency of the body (teeth), it will vibrate with that

resonance as the amplitude of the vibration will be greater as the interference between

the waves is a constructive overlap. When the particles are directed toward the outer

magnetic field, and when the resonance frequency is similar to the frequency of the wave,

the particles absorb those frequencies and thus play a role in the transmission of

infection and thus erosion of the enamel layer, and therefore can be re-mineralization of

the affected parts according to reversible processes.

“Although the precise functions of the amelogenin proteins in enamel formation are not

well defined, these proteins constitute 90% of the enamel organic matrix” [8].

Therefore, interaction of the stearin with the proteins that mineralize the enamel in an

early period (during the secretion of these proteins), which forms a reaction of combustion, and thus consists of plaque, and necrosis before the mineralization of these

proteins, especially when cleaning between teeth, when scratched the fabric of the layer

of protein, (which is dipped vertically between the teeth of the mills) leads to plaque, and

necrosis. Since the enamel of the teeth is also composed of the proteins mentioned

above, they play a role in the reconstruction of the affected enamel layer according to the

interactions of Chiral, and since these proteins are correspond functionally (enantiomer)

to collagen of the cementum, so they play a key role in the reconstruction of damaged

teeth according to chiral process (e.g., by synthesizing complementary molecules of bio

silica to merge with the affected molasses molecules). And because the amino acids are

the basis of protein synthesis, they are considered as enantiomer, and therefore, these

proteins of mineralization react to the processes of Chiral, whether in the case of necrosis

or reconstruction.

In other words, dental entropy exposed to the entropy of the external effect, so necrosis

occurs through the resonance frequency effect. The coupling of entropy can be

eliminated by a certain fractal movement of the target system molecules, but the dental

system is more complex, since the molasses are stable (solid state), and mineralization

proteins may be stimulated to regenerate the affected regions by ultrasonic or infrasonic.

Perhaps teeth play a vital role in appetite, behavior and hair health.

So some studies may be able to find out how to make the bio-process of mineralization of

organic materials to restructure of enamel teeth, which has been damaged by necrosis

without the occurrence of combustion reaction, according to reversible process.

Grant information

The author declared that no grant were involved in supporting this work.

References

1. Rajpar, M. H., Harley, K., Laing, C., Davies, R. M., & Dixon, M. J. (2001).

Mutation of the gene encoding the enamel-specific protein, enamelin, causes

autosomal-dominant amelogenesis imperfecta. Human Molecular Genetics,

10(16), 1673-1677.

2. Zainal, Z., & Yusoff, M. S. A. (1999). Enzymatic interesterification of palm

stearin and palm kernel olein. Journal of the American Oil Chemists’ Society,

76(9), 1003-1008.

3. Weiner, S., & Traub, W. (1986). Organization of hydroxyapatite crystals

within collagen fibrils. FEBS letters, 206(2), 262-266.

4. Jin, Y., & Yip, H. K. (2002). Supragingival calculus: formation and control.

Critical reviews in oral biology & medicine, 13(5), 426-441.

5. Deutsch, D., Palmon, A., Fisher, L. W., Kolodny, N., Termine, J. D., & Young,

M. F. (1991). Sequencing of bovine enamelin (" tuftelin") a novel acidic

enamel protein. Journal of Biological Chemistry, 266(24), 16021-16028.

6. Cashman, M. W., & Sloan, S. B. (2010). Nutrition and nail disease. Clinics in

dermatology, 28(4), 420-425.

7. Jin, Y., & Yip, H. K. (2002). Supragingival calculus: formation and control.

Critical reviews in oral biology & medicine, 13(5), 426-441.

8. Gibson, C. W., Yuan, Z. A., Hall, B., Longenecker, G., Chen, E., Thyagarajan,

T., ... & Harrison, G. (2001). Amelogenin-deficient mice display an

amelogenesis imperfecta phenotype. Journal of Biological Chemistry, 276(34),

31871-31875.