IntroductionThe folliculitis decalvans (FD) is a cicatricial alopecia thought to result from a neutrophilic immune reaction to microbial biofilms, presenting clinically with perifollicular pustules, crusting, and tufted follicular papules (1). On the other hand, lichen planopilaris (LPP) is a T cell–mediated autoimmune disorder—primarily involving CD8⁺ lymphocytes—that targets vulnerable hair follicles with reduced peroxisome proliferator-activated receptor gamma (PPAR-γ) activity. It typically appears as perifollicular erythema and scaling (2).Although FD and LPP are distinct entities, they may share similar features. Morais et al described a series of 13 patients diagnosed with “LPP with pustules” based on the clinical presentation of perifollicular scaling, erythema, crusts, follicular tufts, and pustules (3). The presence of overlapping or sequential findings—whether clinical, dermoscopic, or histopathologic—can complicate the diagnostic process. So the term Folliculitis decalvans and lichen planopilaris phenotypic spectrum (FDLPPs) has been introduced to describe this dual inflammatory presentation, which reflects both a breakdown in immune privilege and microbial imbalance (4). This disease may follow a biphasic pattern, though in some cases features of both alopecia may be observed simultaneously (5).In this case report, we describe a patient who exhibited combined clinical and histological characteristics of both FD and LPP, consistent with the FDLPPs spectrum. Through a detailed analysis of clinical signs, trichoscopic patterns, and histopathological findings, we aim to shed light on the diagnostic challenges and therapeutic considerations in this emerging form of cicatricial alopecia.